Free T3 (Triiodothyronine)

Iron Saturation

WBC (White Blood Cell Count)

Tg Ab (Thyroglobulin Antibodies)

Glucose

TNF-α (Tumor Necrosis Factor-alpha)

Ceruloplasmin

GGT (Gamma-Glutamyl Transferase)

Albumin

Sed Rate (Erythrocyte Sedimentation Rate)

IL-6 (Interleukin-6)

Monocytes (Absolute)

NRBC (Nucleated Red Blood Cells)

25(OH)D (25-Hydroxyvitamin D)

Serum Iron

Uric Acid

A/G Ratio (Albumin/Globulin Ratio)

Fasting Insulin

Lactic Acid

BUN/Creatinine Ratio

Serum Cortisol

Cystatin C

Platelet Count

Ferritin

Total Cholesterol

Sodium

LDL Particle Number

TIBC (Total Iron Binding Capacity)

Triglycerides

Total Testosterone

Calcium

AST (Aspartate Aminotransferase)

LDL Cholesterol (calculated)

Phosphorous

IGF-1 (Insulin-Like Growth Factor 1)

TPO Ab (Thyroid Peroxidase Antibodies)

RDW (Red Cell Distribution Width)

Potassium

Bilirubin (Total and Direct)

HS-CRP (High-Sensitivity C-Reactive Protein)

"Discover the impact of Reverse T3 (rT3) as a biomarker for longevity. Learn how monitoring rT3 levels can optimize health and extend lifespan."

Reverse T3 (rT3)

Reverse T3 (rT3) is a biomarker that has gained attention in longevity research. It is a non-active form of the thyroid hormone T3, and high levels of rT3 have been associated with metabolic dysfunction and chronic stress, both of which are known to contribute to aging. Tracking rT3 levels can provide insight into thyroid function and overall metabolic health, making it a valuable biomarker for assessing longevity. By monitoring and optimizing rT3 levels, individuals can potentially support their overall health and potentially slow down the aging process. Overall, rT3 is a promising biomarker for longevity research and personalized health interventions.

Biomarker Explained

Reverse T3 (rT3) is a biomarker that has gained attention in longevity research due to its ability to provide valuable insight into thyroid function and overall metabolic health. As a non-active form of the thyroid hormone T3, high levels of rT3 have been associated with metabolic dysfunction and chronic stress, both of which are known to contribute to aging. Therefore, tracking rT3 levels can indicate potential issues in thyroid function and metabolic health, which are crucial factors in the aging process. In order to interpret the significance of rT3 levels, it is essential to understand the relationship between rT3 and aging. High levels of rT3 can indicate an imbalance in thyroid function, which can lead to metabolic dysfunction and chronic stress, ultimately contributing to the aging process. Therefore, individuals with elevated rT3 levels may benefit from interventions aimed at optimizing their thyroid function and reducing chronic stress in order to support their overall health and potentially slow down the aging process. Overall, rT3 is a promising biomarker for longevity research and personalized health interventions, as monitoring and optimizing rT3 levels can provide important insights into thyroid function and metabolic health, ultimately contributing to efforts to support longevity and healthy aging.

Keywords:

Reverse T3, rT3, biomarker, thyroid function, metabolic health, aging, longevity

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How does Rapaymcin work?

Rapamycin slows aging by targeting the mTOR pathway, shifting the body’s focus from growth to repair. It promotes cellular recycling, reduces overgrowth linked to disease, and enhances resilience to stress.

Imagine your body as a city, bustling with activity.

Cells are the workers, and mTOR (mechanistic target of rapamycin) is the city planner, deciding where to focus resources – building new structures, cleaning up waste, or repairing old ones.

As we age, mTOR often prioritizes building (cell growth) over maintenance (cellular repair), leading to “clutter” in our bodies that contributes to aging and disease.

This is where Rapamycin comes in.

It acts like a wise advisor to mTOR, convincing it to slow down unnecessary growth projects and focus on clean up and repair instead.

Specifically, Rapamycin:

Activates cellular recycling (autophagy):

Think of autophagy as the city’s waste management system. Damaged parts of cells are broken down and reused, keeping the system efficient and healthy.

Reduces harmful overgrowth:

Overactive mTOR has been linked to diseases such as cancer, cardiovascular disease, and neurodegenerative conditions like Alzheimer’s. By dialing back excessive growth signals, Rapamycin helps prevent these issues.

Supports stress resilience:

When cells are less focused on growing, they’re better equipped to handle stress, repair damage, and maintain long-term health.