Apolipoprotein B

Hemoglobin

LDL Cholesterol (calculated)

DHA (Docosahexaenoic Acid)

ApoA/ApoB Ratio

Free T4 (Thyroxine)

Platelet Count

MCV (Mean Corpuscular Volume)

Neutrophils (Absolute)

MCH (Mean Corpuscular Hemoglobin)

Cystatin C

Lipoprotein(a) [Lp(a)]

RBC Magnesium

Fasting Insulin

Chloride

A/G Ratio (Albumin/Globulin Ratio)

Free T3 (Triiodothyronine)

Uric Acid

MCHC (Mean Corpuscular Hemoglobin Concentration)

Lymphocytes (Absolute)

VLDL Cholesterol (calculated)

UIBC (Unsaturated Iron Binding Capacity)

WBC (White Blood Cell Count)

Fibrinogen

EPA (Eicosapentaenoic Acid)

Sed Rate (Erythrocyte Sedimentation Rate)

Triglycerides

Serum Cortisol

Albumin

Serum Iron

DHEA-S (Dehydroepiandrosterone Sulfate)

HS-CRP (High-Sensitivity C-Reactive Protein)

Tg Ab (Thyroglobulin Antibodies)

Total Cholesterol

Apolipoprotein A1

Eosinophils (Absolute)

Bilirubin (Total and Direct)

Basophils (Absolute)

LDL Particle Number

Hematocrit

Discover the significance of Sed Rate (Erythrocyte Sedimentation Rate) as a biomarker for longevity and overall health. Find out more here.

Sed Rate (Erythrocyte Sedimentation Rate)

Sed Rate (Erythrocyte Sedimentation Rate) is a biomarker commonly used in longevity research to measure inflammation in the body. High levels of inflammation are associated with various age-related diseases and conditions, making Sed Rate a valuable indicator for assessing overall health and potential lifespan. By monitoring and tracking changes in Sed Rate over time, researchers and healthcare professionals can gain valuable insights into the aging process and develop targeted interventions to promote longevity. Additionally, Sed Rate can also be used to assess the efficacy of lifestyle modifications and medical treatments aimed at reducing inflammation and improving overall healthspan.

Biomarker Explained

Sed Rate, also known as Erythrocyte Sedimentation Rate, is a valuable biomarker in longevity research for assessing inflammation in the body. High levels of inflammation are indicative of various age-related diseases and conditions, which can impact overall health and potential lifespan. When interpreting Sed Rate, it is important to consider the individual’s baseline level and monitor changes over time. An increase in Sed Rate may signal heightened inflammation and potential health concerns, while a decrease may indicate successful intervention or a positive response to lifestyle modifications or medical treatments aimed at reducing inflammation. In the context of longevity research, tracking changes in Sed Rate can provide valuable insights into the aging process and help identify targeted interventions for promoting longevity. By using Sed Rate as an indicator for assessing overall health and potential lifespan, researchers and healthcare professionals can develop personalized strategies to improve healthspan and potentially extend lifespan. Overall, Sed Rate serves as a useful tool for assessing the efficacy of interventions aimed at reducing inflammation and improving overall health. Its role in longevity research underscores the importance of understanding and monitoring inflammation as a key factor in promoting healthy aging.

Keywords:

Sed Rate, Erythrocyte Sedimentation Rate, Biomarker, Inflammation, Age-related diseases, Longevity research, Healthspan

How does Rapaymcin work?

Rapamycin slows aging by targeting the mTOR pathway, shifting the body’s focus from growth to repair. It promotes cellular recycling, reduces overgrowth linked to disease, and enhances resilience to stress.

Imagine your body as a city, bustling with activity.

Cells are the workers, and mTOR (mechanistic target of rapamycin) is the city planner, deciding where to focus resources – building new structures, cleaning up waste, or repairing old ones.

As we age, mTOR often prioritizes building (cell growth) over maintenance (cellular repair), leading to “clutter” in our bodies that contributes to aging and disease.

This is where Rapamycin comes in.

It acts like a wise advisor to mTOR, convincing it to slow down unnecessary growth projects and focus on cleanup and repair instead.

Specifically, Rapamycin:

  • Activates cellular recycling (autophagy):
    Think of autophagy as the city’s waste management system. Damaged parts of cells are broken down and reused, keeping the system efficient and healthy.

  • Reduces harmful overgrowth:
    Overactive mTOR has been linked to diseases such as cancer, cardiovascular disease, and neurodegenerative conditions like Alzheimer’s. By dialing back excessive growth signals, Rapamycin helps prevent these issues.

  • Supports stress resilience:
    When cells are less focused on growing, they’re better equipped to handle stress, repair damage, and maintain long-term health.