UIBC (Unsaturated Iron Binding Capacity)

Platelet Count

Reverse T3 (rT3)

Sed Rate (Erythrocyte Sedimentation Rate)

TPO Ab (Thyroid Peroxidase Antibodies)

MCV (Mean Corpuscular Volume)

IGF-1 (Insulin-Like Growth Factor 1)

Triglycerides

Ferritin

RBC (Red Blood Cell Count)

Apolipoprotein B

eGFR (Estimated Glomerular Filtration Rate)

Vitamin A (Retinol)

ALT (Alanine Aminotransferase)

Bilirubin (Total and Direct)

Tg Ab (Thyroglobulin Antibodies)

Creatinine

ANA (Antinuclear Antibody)

BUN/Creatinine Ratio

HOMA-IR (Homeostatic Model Assessment of Insulin Resistance)

VLDL Cholesterol (calculated)

Lymphocytes (Absolute)

Glucose

DHEA-S (Dehydroepiandrosterone Sulfate)

Free T4 (Thyroxine)

Basophils (Absolute)

Hemoglobin A1C

Potassium

Insulin

Ceruloplasmin

Fibrinogen

LDL Particle Size

Free T3 (Triiodothyronine)

Iron Saturation

AST (Aspartate Aminotransferase)

Lipoprotein(a) [Lp(a)]

RDW (Red Cell Distribution Width)

LDL Cholesterol (calculated)

LDH (Lactate Dehydrogenase)

Bicarbonate

Phosphorous, a key biomarker for longevity, plays a vital role in cellular metabolism and energy production, with its levels reflecting overall health and aging.

Phosphorous

Biomarkers play a crucial role in assessing longevity, as they provide valuable insights into the aging process and overall health. Common biomarkers used for longevity purposes include telomere length, which is an indicator of cellular aging, and levels of inflammation markers such as C-reactive protein and interleukin-6. Other important biomarkers include insulin sensitivity, oxidative stress markers, and lipid profiles. By measuring these biomarkers, researchers and healthcare professionals can gain a better understanding of an individual’s biological age and identify potential risk factors for age-related diseases. This information can then be used to develop personalized interventions aimed at promoting healthy aging and extending lifespan.

Biomarker Explained

Biomarkers are essential tools in assessing longevity and overall health. When interpreting biomarkers such as telomere length, levels of inflammation markers, insulin sensitivity, oxidative stress markers, and lipid profiles, it’s important to understand their significance in the aging process. Telomere length, for example, serves as an indicator of cellular aging, with shorter telomeres suggesting more advanced biological age. Elevated levels of inflammation markers such as C-reactive protein and interleukin-6 are indicative of chronic inflammation, which is a key factor in age-related diseases. Insulin sensitivity is a crucial biomarker as it reflects the body’s ability to regulate blood sugar levels and is linked to metabolic health. Oxidative stress markers, such as reactive oxygen species and antioxidant enzymes, provide insights into cellular damage and aging. Lipid profiles, including cholesterol and triglyceride levels, are important biomarkers for cardiovascular health and overall longevity. By interpreting these biomarkers, healthcare professionals and researchers can assess an individual’s biological age, identify potential risk factors for age-related diseases, and develop personalized interventions aimed at promoting healthy aging and increasing lifespan.

Keywords:

telomere length, inflammation markers, insulin sensitivity, oxidative stress markers, lipid profiles, biological age, age-related diseases

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How does Rapaymcin work?

Rapamycin slows aging by targeting the mTOR pathway, shifting the body’s focus from growth to repair. It promotes cellular recycling, reduces overgrowth linked to disease, and enhances resilience to stress.

Imagine your body as a city, bustling with activity.

Cells are the workers, and mTOR (mechanistic target of rapamycin) is the city planner, deciding where to focus resources – building new structures, cleaning up waste, or repairing old ones.

As we age, mTOR often prioritizes building (cell growth) over maintenance (cellular repair), leading to “clutter” in our bodies that contributes to aging and disease.

This is where Rapamycin comes in.

It acts like a wise advisor to mTOR, convincing it to slow down unnecessary growth projects and focus on clean up and repair instead.

Specifically, Rapamycin:

Activates cellular recycling (autophagy):

Think of autophagy as the city’s waste management system. Damaged parts of cells are broken down and reused, keeping the system efficient and healthy.

Reduces harmful overgrowth:

Overactive mTOR has been linked to diseases such as cancer, cardiovascular disease, and neurodegenerative conditions like Alzheimer’s. By dialing back excessive growth signals, Rapamycin helps prevent these issues.

Supports stress resilience:

When cells are less focused on growing, they’re better equipped to handle stress, repair damage, and maintain long-term health.