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Sed Rate (Erythrocyte Sedimentation Rate)

Potassium

Apolipoprotein A1

Total Testosterone

TNF-α (Tumor Necrosis Factor-alpha)

Chloride

Copper Serum

MCV (Mean Corpuscular Volume)

Serum Cortisol

GGT (Gamma-Glutamyl Transferase)

LDL Cholesterol (calculated)

ANA (Antinuclear Antibody)

ApoA/ApoB Ratio

HDL Cholesterol

Phosphorous

Monocytes (Absolute)

Lactic Acid

Triglycerides

Fibrinogen

eGFR (Estimated Glomerular Filtration Rate)

RBC Magnesium

Fasting Insulin

Hemoglobin

EPA (Eicosapentaenoic Acid)

TIBC (Total Iron Binding Capacity)

DHA (Docosahexaenoic Acid)

Insulin

RDW (Red Cell Distribution Width)

RBC (Red Blood Cell Count)

DHEA-S (Dehydroepiandrosterone Sulfate)

Neutrophils (Absolute)

Glucose

Hematocrit

AST (Aspartate Aminotransferase)

LDH (Lactate Dehydrogenase)

MCH (Mean Corpuscular Hemoglobin)

Bicarbonate

IGF-1 (Insulin-Like Growth Factor 1)

Ceruloplasmin

Tg Ab (Thyroglobulin Antibodies)
Fasting Insulin, a biomarker for longevity, can indicate risk for diabetes and metabolic syndrome. Managing levels may support healthy aging.

Fasting Insulin

Fasting Insulin is a crucial biomarker for assessing longevity. Elevated levels of fasting insulin are associated with insulin resistance, a precursor to type 2 diabetes and other chronic diseases. Maintaining optimal fasting insulin levels through a balanced diet and regular physical activity is essential for promoting longevity and overall health. As a longevity expert, I recommend monitoring fasting insulin levels as part of a comprehensive biomarker panel to evaluate metabolic health and guide personalized interventions for healthy aging. By addressing imbalances in fasting insulin, individuals can optimize their longevity potential and decrease the risk of age-related chronic diseases.

Biomarker Explained

Fasting insulin is a critical biomarker for assessing longevity and metabolic health. Elevated levels of fasting insulin are indicative of insulin resistance, a condition that precedes the development of type 2 diabetes and other chronic diseases. Monitoring fasting insulin levels is essential for promoting longevity and overall health. A balanced diet and regular physical activity play pivotal roles in maintaining optimal fasting insulin levels. Through personalized interventions, individuals can address imbalances in fasting insulin to optimize their longevity potential and reduce the risk of age-related chronic diseases. As a longevity expert, I recommend integrating monitoring of fasting insulin levels into a comprehensive biomarker panel to assess metabolic health and guide personalized interventions for healthy aging. By doing so, individuals can take proactive steps to support their longevity and well-being.

Keywords:

Fasting insulin, longevity, metabolic health, insulin resistance, type 2 diabetes, chronic diseases, personalized interventions

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Prescription products require an online consultation with a physician who will determine if a prescription is appropriate. See product page for important safety information. Benefits outlined on Longevity Direct are based on 3rd party studies.

Note: The above statements have not been evaluated by the Food and Drug Administration. These products are not intended to diagnose, treat, cure, or prevent any disease. If you are in need of emergency medical attention, please call 911 immediately.

Longevity Direct logo in color – promoting personalized longevity insights and AI-driven health optimization

Rapamycin

Metformin

GLP-1

General Longevity
Heart Health
Weight Management
Glucose Control
Cognitive Function
Diabetes Prevention
Mood Support
Energy & Fatigue
Autoimmune Support
Blood Pressure
Chronic Pain & Arthritis
Aging Skin
Men’s Aging
Women’s Aging

Longevity Library
About

get Started
Client portal

Contact

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How does Rapaymcin work?

Rapamycin slows aging by targeting the mTOR pathway, shifting the body’s focus from growth to repair. It promotes cellular recycling, reduces overgrowth linked to disease, and enhances resilience to stress.

Imagine your body as a city, bustling with activity.

Cells are the workers, and mTOR (mechanistic target of rapamycin) is the city planner, deciding where to focus resources – building new structures, cleaning up waste, or repairing old ones.

As we age, mTOR often prioritizes building (cell growth) over maintenance (cellular repair), leading to “clutter” in our bodies that contributes to aging and disease.

This is where Rapamycin comes in.

It acts like a wise advisor to mTOR, convincing it to slow down unnecessary growth projects and focus on clean up and repair instead.

Specifically, Rapamycin:

Activates cellular recycling (autophagy):

Think of autophagy as the city’s waste management system. Damaged parts of cells are broken down and reused, keeping the system efficient and healthy.

Reduces harmful overgrowth:

Overactive mTOR has been linked to diseases such as cancer, cardiovascular disease, and neurodegenerative conditions like Alzheimer’s. By dialing back excessive growth signals, Rapamycin helps prevent these issues.

Supports stress resilience:

When cells are less focused on growing, they’re better equipped to handle stress, repair damage, and maintain long-term health.