VLDL Cholesterol (calculated)

Total Protein

Monocytes (Absolute)

Serum Cortisol

Total Testosterone

IL-6 (Interleukin-6)

Hematocrit

HS-CRP (High-Sensitivity C-Reactive Protein)

BUN (Blood Urea Nitrogen)

Apolipoprotein A1

Chloride

MCV (Mean Corpuscular Volume)

Sed Rate (Erythrocyte Sedimentation Rate)

Vitamin A (Retinol)

Alkaline Phosphatase (ALP)

Platelet Count

TNF-α (Tumor Necrosis Factor-alpha)

25(OH)D (25-Hydroxyvitamin D)

RBC (Red Blood Cell Count)

LDH (Lactate Dehydrogenase)

SHBG (Sex Hormone Binding Globulin)

Neutrophils (Absolute)

TIBC (Total Iron Binding Capacity)

Lactic Acid

Phosphorous

Bicarbonate

BUN/Creatinine Ratio

Albumin

GGT (Gamma-Glutamyl Transferase)

Potassium

Uric Acid

UIBC (Unsaturated Iron Binding Capacity)

Apolipoprotein B

MCH (Mean Corpuscular Hemoglobin)

DHEA-S (Dehydroepiandrosterone Sulfate)

Serum Iron

HOMA-IR (Homeostatic Model Assessment of Insulin Resistance)

Copper Serum

Hemoglobin A1C

Lymphocytes (Absolute)

Lipoprotein(a) [Lp(a)] is a biomarker used in longevity research to assess cardiovascular health and risk of developing heart disease.

Lipoprotein(a) [Lp(a)]

Lipoprotein(a) [Lp(a)] is a biomarker that has been increasingly recognized for its potential role in predicting longevity. Elevated levels of Lp(a) have been linked to an increased risk of cardiovascular diseases, which are known to impact overall life expectancy. Monitoring Lp(a) levels can provide valuable insights into an individual’s risk for developing age-related health issues and mortality. As part of a comprehensive longevity assessment, measuring and managing Lp(a) levels can be a crucial component in identifying and mitigating potential risk factors for decreased lifespan. Understanding the impact of Lp(a) on longevity can guide personalized interventions to promote healthy aging and extended lifespan.

Biomarker Explained

Lipoprotein(a) (Lp(a)) is a biomarker that has garnered increasing attention due to its potential role in predicting longevity. Elevated levels of Lp(a) have been associated with a higher risk of developing cardiovascular diseases, which are known to impact overall life expectancy. Therefore, monitoring Lp(a) levels can provide valuable insights into an individual’s risk for age-related health issues and mortality. In the context of a comprehensive longevity assessment, the measurement and management of Lp(a) levels can be a critical component in identifying and addressing potential risk factors for decreased lifespan. Understanding the influence of Lp(a) on longevity can help guide personalized interventions to promote healthy aging and extend lifespan. As such, integrating the assessment of Lp(a) levels into longevity evaluations can aid in identifying individuals who may benefit from targeted interventions to mitigate the impact of elevated Lp(a) on their overall lifespan.

Keywords:

Lipoprotein(a), Lp(a), biomarker, longevity, cardiovascular diseases, risk factors, personalized interventions

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How does Rapaymcin work?

Rapamycin slows aging by targeting the mTOR pathway, shifting the body’s focus from growth to repair. It promotes cellular recycling, reduces overgrowth linked to disease, and enhances resilience to stress.

Imagine your body as a city, bustling with activity.

Cells are the workers, and mTOR (mechanistic target of rapamycin) is the city planner, deciding where to focus resources – building new structures, cleaning up waste, or repairing old ones.

As we age, mTOR often prioritizes building (cell growth) over maintenance (cellular repair), leading to “clutter” in our bodies that contributes to aging and disease.

This is where Rapamycin comes in.

It acts like a wise advisor to mTOR, convincing it to slow down unnecessary growth projects and focus on clean up and repair instead.

Specifically, Rapamycin:

Activates cellular recycling (autophagy):

Think of autophagy as the city’s waste management system. Damaged parts of cells are broken down and reused, keeping the system efficient and healthy.

Reduces harmful overgrowth:

Overactive mTOR has been linked to diseases such as cancer, cardiovascular disease, and neurodegenerative conditions like Alzheimer’s. By dialing back excessive growth signals, Rapamycin helps prevent these issues.

Supports stress resilience:

When cells are less focused on growing, they’re better equipped to handle stress, repair damage, and maintain long-term health.