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Glucose

Insulin

Tg Ab (Thyroglobulin Antibodies)

IGF-1 (Insulin-Like Growth Factor 1)

Total Protein

Uric Acid

Iron Saturation

Platelet Count

TNF-α (Tumor Necrosis Factor-alpha)

MCHC (Mean Corpuscular Hemoglobin Concentration)

GGT (Gamma-Glutamyl Transferase)

MCV (Mean Corpuscular Volume)

TIBC (Total Iron Binding Capacity)

TSH (Thyroid Stimulating Hormone)

ApoA/ApoB Ratio

Apolipoprotein A1

Creatinine

eGFR (Estimated Glomerular Filtration Rate)

Bilirubin (Total and Direct)

UIBC (Unsaturated Iron Binding Capacity)

Hemoglobin

Monocytes (Absolute)

Chloride

25(OH)D (25-Hydroxyvitamin D)

Triglycerides

Eosinophils (Absolute)

Albumin

Immature Granulocytes

Free T3 (Triiodothyronine)

Free T4 (Thyroxine)

Serum Cortisol

RBC Magnesium

Bicarbonate

Total Testosterone

VLDL Cholesterol (calculated)

LDH (Lactate Dehydrogenase)

Fasting Insulin

Hemoglobin A1C

Vitamin A (Retinol)

HS-CRP (High-Sensitivity C-Reactive Protein)
ALP is a biomarker commonly used for assessing longevity. Maintaining optimal levels of this enzyme can be a key indicator of overall health and longevity.

Alkaline Phosphatase (ALP)

Alkaline Phosphatase (ALP) is a biomarker that is often used in longevity studies to assess overall health and potential lifespan. ALP levels in the blood can indicate liver or bone health, as well as other metabolic processes. High ALP levels may suggest underlying liver disease or bone disorders, while low levels can be associated with malnutrition or certain genetic conditions. Monitoring ALP as part of a comprehensive biomarker panel can provide valuable insights into an individual’s overall health and potential longevity, helping to identify areas for intervention and personalized health optimization strategies.

Biomarker Explained

Alkaline Phosphatase (ALP) is a crucial biomarker utilized in longevity studies to evaluate an individual’s overall health and potential lifespan. ALP levels in the blood can yield valuable information regarding liver and bone health, as well as other metabolic processes. Elevated ALP levels may indicate underlying liver disease or bone disorders, necessitating further investigation and potential intervention. On the other hand, low ALP levels can be associated with malnutrition or certain genetic conditions, highlighting the need for personalized health optimization strategies. Monitoring ALP as part of a comprehensive biomarker panel can offer insightful data on an individual’s overall health and potential longevity, enabling the identification of areas for intervention and the development of personalized health strategies. This underscores the importance of incorporating ALP into longevity studies and personalized health plans to maximize overall well-being and longevity potential.

Keywords:

Alkaline Phosphatase, ALP, Biomarker, Longevity, Liver health, Bone health, Personalized health strategies

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Longevity Direct logo in color – promoting personalized longevity insights and AI-driven health optimization

Rapamycin

Metformin

GLP-1

General Longevity
Heart Health
Weight Management
Glucose Control
Cognitive Function
Diabetes Prevention
Mood Support
Energy & Fatigue
Autoimmune Support
Blood Pressure
Chronic Pain & Arthritis
Aging Skin
Men’s Aging
Women’s Aging

Longevity Library
About

get Started
Client portal

Contact

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How does Rapaymcin work?

Rapamycin slows aging by targeting the mTOR pathway, shifting the body’s focus from growth to repair. It promotes cellular recycling, reduces overgrowth linked to disease, and enhances resilience to stress.

Imagine your body as a city, bustling with activity.

Cells are the workers, and mTOR (mechanistic target of rapamycin) is the city planner, deciding where to focus resources – building new structures, cleaning up waste, or repairing old ones.

As we age, mTOR often prioritizes building (cell growth) over maintenance (cellular repair), leading to “clutter” in our bodies that contributes to aging and disease.

This is where Rapamycin comes in.

It acts like a wise advisor to mTOR, convincing it to slow down unnecessary growth projects and focus on clean up and repair instead.

Specifically, Rapamycin:

Activates cellular recycling (autophagy):

Think of autophagy as the city’s waste management system. Damaged parts of cells are broken down and reused, keeping the system efficient and healthy.

Reduces harmful overgrowth:

Overactive mTOR has been linked to diseases such as cancer, cardiovascular disease, and neurodegenerative conditions like Alzheimer’s. By dialing back excessive growth signals, Rapamycin helps prevent these issues.

Supports stress resilience:

When cells are less focused on growing, they’re better equipped to handle stress, repair damage, and maintain long-term health.