Hemoglobin A1C

Free T3 (Triiodothyronine)

Creatinine

Serum Iron

Hemoglobin

eGFR (Estimated Glomerular Filtration Rate)

IGF-1 (Insulin-Like Growth Factor 1)

Iron Saturation

HOMA-IR (Homeostatic Model Assessment of Insulin Resistance)

RBC (Red Blood Cell Count)

Eosinophils (Absolute)

SHBG (Sex Hormone Binding Globulin)

WBC (White Blood Cell Count)

HS-CRP (High-Sensitivity C-Reactive Protein)

BUN/Creatinine Ratio

Free T4 (Thyroxine)

Serum Cortisol

Apolipoprotein B

Apolipoprotein A1

MCHC (Mean Corpuscular Hemoglobin Concentration)

LDH (Lactate Dehydrogenase)

HDL Cholesterol

TIBC (Total Iron Binding Capacity)

Lipoprotein(a) [Lp(a)]

Bilirubin (Total and Direct)

Sed Rate (Erythrocyte Sedimentation Rate)

Bicarbonate

LDL Cholesterol (calculated)

Potassium

Fibrinogen

Platelet Count

BUN (Blood Urea Nitrogen)

LDL Particle Number

ApoA/ApoB Ratio

Insulin

Fasting Insulin

Free Testosterone

DHA (Docosahexaenoic Acid)

NRBC (Nucleated Red Blood Cells)

DHEA-S (Dehydroepiandrosterone Sulfate)

"Learn about the significance of iron saturation as a biomarker for longevity and overall health. Discover how monitoring this marker can impact your lifespan."

Iron Saturation

Iron saturation is a key biomarker that longevity experts use to assess overall health and potential longevity. This biomarker measures the amount of iron that is bound to transferrin in the blood, indicating the body’s ability to transport and utilize iron effectively. Maintaining an optimal iron saturation level is crucial for longevity, as both iron deficiency and excess can lead to various health issues and shorten lifespan. By monitoring and managing iron saturation levels, individuals can support healthy aging and reduce the risk of age-related diseases. Longevity experts utilize this biomarker as part of a comprehensive approach to promoting longevity and vitality.

Biomarker Explained

Iron saturation is a critical biomarker used by longevity experts to evaluate overall health and potential longevity. It measures the amount of iron bound to transferrin in the blood, reflecting the body’s ability to transport and utilize iron effectively. An optimal iron saturation level is essential for promoting longevity, as both iron deficiency and excess can lead to various health issues and shorten lifespan. Interpreting iron saturation involves assessing whether the level falls within the optimal range, typically between 20-50%. A lower iron saturation may indicate iron deficiency, which can lead to fatigue, weakened immune function, and cognitive impairment. Conversely, high iron saturation levels can result in oxidative stress and tissue damage, increasing the risk of age-related diseases such as heart disease and cancer. By monitoring and managing iron saturation levels, individuals can support healthy aging and reduce the risk of age-related diseases. Longevity experts use this biomarker as part of a comprehensive approach to promoting longevity and vitality, emphasizing the importance of maintaining an optimal iron saturation level through balanced dietary intake and appropriate supplementation. Regular monitoring of iron saturation enables early intervention to prevent adverse health outcomes and optimize overall well-being.

Keywords:

Iron saturation, transferrin, longevity, biomarker, optimal range, iron deficiency, oxidative stress

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How does Rapaymcin work?

Rapamycin slows aging by targeting the mTOR pathway, shifting the body’s focus from growth to repair. It promotes cellular recycling, reduces overgrowth linked to disease, and enhances resilience to stress.

Imagine your body as a city, bustling with activity.

Cells are the workers, and mTOR (mechanistic target of rapamycin) is the city planner, deciding where to focus resources – building new structures, cleaning up waste, or repairing old ones.

As we age, mTOR often prioritizes building (cell growth) over maintenance (cellular repair), leading to “clutter” in our bodies that contributes to aging and disease.

This is where Rapamycin comes in.

It acts like a wise advisor to mTOR, convincing it to slow down unnecessary growth projects and focus on clean up and repair instead.

Specifically, Rapamycin:

Activates cellular recycling (autophagy):

Think of autophagy as the city’s waste management system. Damaged parts of cells are broken down and reused, keeping the system efficient and healthy.

Reduces harmful overgrowth:

Overactive mTOR has been linked to diseases such as cancer, cardiovascular disease, and neurodegenerative conditions like Alzheimer’s. By dialing back excessive growth signals, Rapamycin helps prevent these issues.

Supports stress resilience:

When cells are less focused on growing, they’re better equipped to handle stress, repair damage, and maintain long-term health.