Ferritin

Ceruloplasmin

HOMA-IR (Homeostatic Model Assessment of Insulin Resistance)

Total Testosterone

BUN/Creatinine Ratio

Insulin

Serum Cortisol

EPA (Eicosapentaenoic Acid)

ANA (Antinuclear Antibody)

NRBC (Nucleated Red Blood Cells)

Potassium

TIBC (Total Iron Binding Capacity)

DHEA-S (Dehydroepiandrosterone Sulfate)

Glucose

Bicarbonate

Basophils (Absolute)

LDH (Lactate Dehydrogenase)

Neutrophils (Absolute)

RBC Magnesium

Total Cholesterol

Calcium

UIBC (Unsaturated Iron Binding Capacity)

Fasting Insulin

eGFR (Estimated Glomerular Filtration Rate)

Eosinophils (Absolute)

Bilirubin (Total and Direct)

TPO Ab (Thyroid Peroxidase Antibodies)

Serum Iron

HDL Cholesterol

Uric Acid

Vitamin A (Retinol)

Tg Ab (Thyroglobulin Antibodies)

Hemoglobin A1C

Monocytes (Absolute)

Platelet Count

RBC (Red Blood Cell Count)

Cystatin C

A/G Ratio (Albumin/Globulin Ratio)

HS-CRP (High-Sensitivity C-Reactive Protein)

IGF-1 (Insulin-Like Growth Factor 1)

Ensure optimal longevity with biomarker testing. Homocysteine levels indicate cardiovascular health and can be managed through lifestyle changes.

Homocysteine

Homocysteine is a biomarker commonly used in longevity research due to its association with cardiovascular disease and overall mortality risk. Elevated levels of homocysteine have been linked to increased risk of stroke, heart disease, and cognitive decline. Monitoring homocysteine levels can provide valuable insight into an individual’s potential risk for these age-related health issues. By understanding and managing homocysteine levels, individuals may be able to make lifestyle and dietary changes aimed at reducing their risk of age-related diseases and promoting longevity. As such, homocysteine is a valuable biomarker in the pursuit of extending and improving quality of life.

Biomarker Explained

Homocysteine is a biomarker commonly used in longevity research due to its association with cardiovascular disease and overall mortality risk. Elevated levels of homocysteine have been linked to increased risk of stroke, heart disease, and cognitive decline. In interpreting homocysteine levels, it’s important to note that optimal levels may vary based on age, sex, and other individual factors. Generally, levels below 10 micromoles per liter (μmol/L) are considered normal, while levels between 10-15 μmol/L are considered borderline and levels above 15 μmol/L are considered high. Monitoring homocysteine levels can provide valuable insight into an individual’s potential risk for these age-related health issues. It’s also important to consider other factors that can affect homocysteine levels, such as diet, lifestyle, and certain medications. By understanding and managing homocysteine levels, individuals may be able to make lifestyle and dietary changes aimed at reducing their risk of age-related diseases and promoting longevity. This could include increasing intake of B vitamins (such as B6, B12, and folate), which are known to help lower homocysteine levels. In summary, interpreting homocysteine levels is an important aspect of longevity research and can provide valuable information for individuals looking to improve their quality of life and reduce their risk of age-related diseases.

Keywords:

Homocysteine, longevity research, cardiovascular disease, mortality risk, stroke, heart disease, cognitive decline, B vitamins

How does Rapaymcin work?

Rapamycin slows aging by targeting the mTOR pathway, shifting the body’s focus from growth to repair. It promotes cellular recycling, reduces overgrowth linked to disease, and enhances resilience to stress.

Imagine your body as a city, bustling with activity.

Cells are the workers, and mTOR (mechanistic target of rapamycin) is the city planner, deciding where to focus resources – building new structures, cleaning up waste, or repairing old ones.

As we age, mTOR often prioritizes building (cell growth) over maintenance (cellular repair), leading to “clutter” in our bodies that contributes to aging and disease.

This is where Rapamycin comes in.

It acts like a wise advisor to mTOR, convincing it to slow down unnecessary growth projects and focus on clean up and repair instead.

Specifically, Rapamycin:

Activates cellular recycling (autophagy):

Think of autophagy as the city’s waste management system. Damaged parts of cells are broken down and reused, keeping the system efficient and healthy.

Reduces harmful overgrowth:

Overactive mTOR has been linked to diseases such as cancer, cardiovascular disease, and neurodegenerative conditions like Alzheimer’s. By dialing back excessive growth signals, Rapamycin helps prevent these issues.

Supports stress resilience:

When cells are less focused on growing, they’re better equipped to handle stress, repair damage, and maintain long-term health.