Serum Cortisol

Immature Granulocytes

Lipoprotein(a) [Lp(a)]

LDL Cholesterol (calculated)

TSH (Thyroid Stimulating Hormone)

EPA (Eicosapentaenoic Acid)

UIBC (Unsaturated Iron Binding Capacity)

LDH (Lactate Dehydrogenase)

MCV (Mean Corpuscular Volume)

BUN/Creatinine Ratio

DHEA-S (Dehydroepiandrosterone Sulfate)

Bilirubin (Total and Direct)

MCHC (Mean Corpuscular Hemoglobin Concentration)

Neutrophils (Absolute)

Alkaline Phosphatase (ALP)

ALT (Alanine Aminotransferase)

Sodium

Basophils (Absolute)

HS-CRP (High-Sensitivity C-Reactive Protein)

HOMA-IR (Homeostatic Model Assessment of Insulin Resistance)

Vitamin A (Retinol)

HDL Cholesterol

LDL Particle Size

VLDL Cholesterol (calculated)

Ferritin

Hemoglobin

Platelet Count

Free T3 (Triiodothyronine)

Creatinine

Eosinophils (Absolute)

Apolipoprotein A1

IL-6 (Interleukin-6)

Tg Ab (Thyroglobulin Antibodies)

Reverse T3 (rT3)

Chloride

DHA (Docosahexaenoic Acid)

A/G Ratio (Albumin/Globulin Ratio)

eGFR (Estimated Glomerular Filtration Rate)

Hemoglobin A1C

Copper Serum

Discover the significance of Sed Rate (Erythrocyte Sedimentation Rate) as a biomarker for longevity and overall health. Find out more here.

Sed Rate (Erythrocyte Sedimentation Rate)

Sed Rate (Erythrocyte Sedimentation Rate) is a biomarker commonly used in longevity research to measure inflammation in the body. High levels of inflammation are associated with various age-related diseases and conditions, making Sed Rate a valuable indicator for assessing overall health and potential lifespan. By monitoring and tracking changes in Sed Rate over time, researchers and healthcare professionals can gain valuable insights into the aging process and develop targeted interventions to promote longevity. Additionally, Sed Rate can also be used to assess the efficacy of lifestyle modifications and medical treatments aimed at reducing inflammation and improving overall healthspan.

Biomarker Explained

Sed Rate, also known as Erythrocyte Sedimentation Rate, is a valuable biomarker in longevity research for assessing inflammation in the body. High levels of inflammation are indicative of various age-related diseases and conditions, which can impact overall health and potential lifespan. When interpreting Sed Rate, it is important to consider the individual’s baseline level and monitor changes over time. An increase in Sed Rate may signal heightened inflammation and potential health concerns, while a decrease may indicate successful intervention or a positive response to lifestyle modifications or medical treatments aimed at reducing inflammation. In the context of longevity research, tracking changes in Sed Rate can provide valuable insights into the aging process and help identify targeted interventions for promoting longevity. By using Sed Rate as an indicator for assessing overall health and potential lifespan, researchers and healthcare professionals can develop personalized strategies to improve healthspan and potentially extend lifespan. Overall, Sed Rate serves as a useful tool for assessing the efficacy of interventions aimed at reducing inflammation and improving overall health. Its role in longevity research underscores the importance of understanding and monitoring inflammation as a key factor in promoting healthy aging.

Keywords:

Sed Rate, Erythrocyte Sedimentation Rate, Biomarker, Inflammation, Age-related diseases, Longevity research, Healthspan

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How does Rapaymcin work?

Rapamycin slows aging by targeting the mTOR pathway, shifting the body’s focus from growth to repair. It promotes cellular recycling, reduces overgrowth linked to disease, and enhances resilience to stress.

Imagine your body as a city, bustling with activity.

Cells are the workers, and mTOR (mechanistic target of rapamycin) is the city planner, deciding where to focus resources – building new structures, cleaning up waste, or repairing old ones.

As we age, mTOR often prioritizes building (cell growth) over maintenance (cellular repair), leading to “clutter” in our bodies that contributes to aging and disease.

This is where Rapamycin comes in.

It acts like a wise advisor to mTOR, convincing it to slow down unnecessary growth projects and focus on clean up and repair instead.

Specifically, Rapamycin:

Activates cellular recycling (autophagy):

Think of autophagy as the city’s waste management system. Damaged parts of cells are broken down and reused, keeping the system efficient and healthy.

Reduces harmful overgrowth:

Overactive mTOR has been linked to diseases such as cancer, cardiovascular disease, and neurodegenerative conditions like Alzheimer’s. By dialing back excessive growth signals, Rapamycin helps prevent these issues.

Supports stress resilience:

When cells are less focused on growing, they’re better equipped to handle stress, repair damage, and maintain long-term health.